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  1. ↑ Freedberg และคณะ (2003). Fitzpatrick's Dermatology in General Medicine (6th ed.). McGraw-Hill. ISBN 0-07-138076-0. Explicit use of et al. in: |authors= (help)CS1 maint: uses authors parameter (link)
  2. ↑ Rapini, Ronald P.; Bolognia, Jean L.; Jorizzo, Joseph L. (2007). Dermatology. St. Louis: Mosby. ISBN 1-4160-2999-0.CS1 maint: uses authors parameter (link)
  3. ↑ "Dermatitis herpetiformis and rheumatoid arthritis". 2002. PMID 17656946. Cite journal requires |journal= (help)
  4. ↑ "Dermatitis Herpetiformis". American Osteopathic College of Dermatology. Archived from the original on 2018-10-29. Unknown parameter |deadurl= ignored (help)
  5. 1 2 3 4 5 6 Fasano, A (April 2005). "Clinical presentation of celiac disease in the pediatric population". Gastroenterology (Review). 128 (4 Suppl 1): S68-73. doi:10.1053/j.gastro.2005.02.015. PMID 15825129.
  6. ↑ "Symptoms & Causes of Celiac Disease | NIDDK". National Institute of Diabetes and Digestive and Kidney Diseases. June 2016. Archived from the original on 2017-04-24. สืบค้นเมื่อ 2017-04-24. Unknown parameter |deadurl= ignored (help)
  7. 1 2 Lebwohl, B; Ludvigsson, JF; Green, PH (October 2015). "Celiac disease and non-celiac gluten sensitivity". BMJ (Review). 351: h4347. doi:10.1136/bmj.h4347. PMC 4596973. PMID 26438584. Celiac disease occurs in about 1% of the population worldwide, although most people with the condition are undiagnosed. It can cause a wide variety of symptoms, both intestinal and extra-intestinal because it is a systemic autoimmune disease that is triggered by dietary gluten. Patients with coeliac disease are at increased risk of cancer, including a twofold to fourfold increased risk of non-Hodgkin’s lymphoma and a more than 30-fold increased risk of small intestinal adenocarcinoma, and they have a 1.4-fold increased risk of death.CS1 maint: uses authors parameter (link)
  8. 1 2 Lundin, KE; Wijmenga, C (September 2015). "Coeliac disease and autoimmune disease-genetic overlap and screening". Nature Reviews. Gastroenterology & Hepatology (Review). 12 (9): 507–15. doi:10.1038/nrgastro.2015.136. PMID 26303674. The abnormal immunological response elicited by gluten-derived proteins can lead to the production of several different autoantibodies, which affect different systems.CS1 maint: uses authors parameter (link)
  9. 1 2 "Celiac disease". World Gastroenterology Organisation Global Guidelines. July 2016. Archived from the original on 2017-03-17. สืบค้นเมื่อ 2017-04-23. Unknown parameter |deadurl= ignored (help)
  10. 1 2 3 4 Ciccocioppo, R; Kruzliak, P; Cangemi, GC; Pohanka, M; Betti, E; Lauret, E; Rodrigo, L (2015-10-22). "The Spectrum of Differences between Childhood and Adulthood Celiac Disease". Nutrients (Review). 7 (10): 8733–51. doi:10.3390/nu7105426. PMC 4632446. PMID 26506381. Several additional studies in extensive series of coeliac patients have clearly shown that TG2A sensitivity varies depending on the severity of duodenal damage, and reaches almost 100% in the presence of complete villous atrophy (more common in children under three years), 70% for subtotal atrophy, and up to 30% when only an increase in IELs is present. (IELs: intraepithelial lymphocytes) CS1 maint: uses authors parameter (link)
  11. ↑ Lionetti, E; Francavilla, R; Pavone, P; Pavone, L; Francavilla, T; Pulvirenti, A; Giugno, R; Ruggieri, M (August 2010). "The neurology of coeliac disease in childhood: what is the evidence? A systematic review and meta-analysis". Developmental Medicine and Child Neurology. 52 (8): 700–7. doi:10.1111/j.1469-8749.2010.03647.x. PMID 20345955.CS1 maint: uses authors parameter (link)
  12. 1 2 3 4 Husby, S; Koletzko, S; Korponay-Szabó, IR; Mearin, ML; Phillips, A; Shamir, R; Troncone, R; Giersiepen, K; Branski, D; Catassi, C; Lelgeman, M; Mäki, M; Ribes-Koninckx, C; Ventura, A; Zimmer, KP; ESPGHAN Working Group on Coeliac Disease Diagnosis; ESPGHAN Gastroenterology Committee; European Society for Pediatric Gastroenterology, Hepatology, Nutrition (January 2012). "European Society for Pediatric Gastroenterology, Hepatology, and Nutrition guidelines for the diagnosis of coeliac disease" (PDF). J Pediatr Gastroenterol Nutr (Practice Guideline). 54 (1): 136–60. doi:10.1097/MPG.0b013e31821a23d0. PMID 22197856. Archived from the original (PDF) on 2016-04-03. Since 1990, the understanding of the pathological processes of CD has increased enormously, leading to a change in the clinical paradigm of CD from a chronic, gluten-dependent enteropathy of childhood to a systemic disease with chronic immune features affecting different organ systems. (...) atypical symptoms may be considerably more common than classic symptoms Unknown parameter |deadurl= ignored (help)CS1 maint: uses authors parameter (link)
  13. 1 2 Tovoli, F; Masi, C; Guidetti, E; Negrini, G; Paterini, P; Bolondi, L (March 2015). "Clinical and diagnostic aspects of gluten related disorders". World Journal of Clinical Cases (Review). 3 (3): 275–84. doi:10.12998/wjcc.v3.i3.275. PMC 4360499. PMID 25789300.CS1 maint: uses authors parameter (link)
  14. 1 2 3 4 5 6 7 "Celiac Disease". NIDDKD. June 2015. Archived from the original on 2016-03-13. สืบค้นเมื่อ 2016-03-17. Unknown parameter |deadurl= ignored (help)
  15. 1 2 Vivas, S; Vaquero, L; Rodríguez-Martín, L; Caminero, A (November 2015). "Age-related differences in celiac disease: Specific characteristics of adult presentation". World Journal of Gastrointestinal Pharmacology and Therapeutics (Review). 6 (4): 207–12. doi:10.4292/wjgpt.v6.i4.207. PMC 4635160. PMID 26558154. In addition, the presence of intraepithelial lymphocytosis and/or villous atrophy and crypt hyperplasia of small-bowel mucosa, and clinical remission after withdrawal of gluten from the diet, are also used for diagnosis antitransglutaminase antibody (tTGA) titers and the degree of histological lesions inversely correlate with age. Thus, as the age of diagnosis increases antibody titers decrease and histological damage is less marked. It is common to find adults without villous atrophy showing only an inflammatory pattern in duodenal mucosa biopsies: Lymphocytic enteritis (Marsh I) or added crypt hyperplasia (Marsh II)CS1 maint: uses authors parameter (link)
  16. ↑ Ferri, Fred F. (2010). Ferri's differential diagnosis : a practical guide to the differential diagnosis of symptoms, signs, and clinical disorders (2nd ed.). Philadelphia, PA: Elsevier/Mosby. p. Chapter C. ISBN 0323076998.
  17. 1 2 See, JA; Kaukinen, K; Makharia, GK; Gibson, PR; Murray, JA (October 2015). "Practical insights into gluten-free diets". Nature Reviews. Gastroenterology & Hepatology (Review). 12 (10): 580–91. doi:10.1038/nrgastro.2015.156. PMID 26392070. A lack of symptoms and/or negative serological markers are not reliable indicators of mucosal response to the diet. Furthermore, up to 30% of patients continue to have gastrointestinal symptoms despite a strict GFD.122,124 If adherence is questioned, a structured interview by a qualified dietitian can help to identify both intentional and inadvertent sources of gluten.CS1 maint: uses authors parameter (link)
  18. 1 2 Fasano, A; Catassi, C (December 2012). "Clinical practice. Celiac disease". The New England Journal of Medicine (Review). 367 (25): 2419–26. doi:10.1056/NEJMcp1113994. PMID 23252527.CS1 maint: uses authors parameter (link)
  19. ↑ บัญชีจำแนกโรคระหว่างประเทศ ฉบับประเทศไทย (อังกฤษ-ไทย) ฉบับปี 2009. สำนักนโยบายและยุทธศาสตร์ สำนักงานปลัดกระทรวงสาธารณสุข, 2552.
  20. ↑ Newnham, Evan D (2017). "Coeliac disease in the 21st century: Paradigm shifts in the modern age". Journal of Gastroenterology and Hepatology. 32: 82–85. doi:10.1111/jgh.13704. PMID 28244672. Presentation of CD with malabsorptive symptoms or malnutrition is now the exception rather than the rule.
  21. 1 2 M, Rostami Nejad; Hogg-Kollars, S; Ishaq, S; Rostami, K (2011). "Subclinical celiac disease and gluten sensitivity". Gastroenterol Hepatol Bed Bench (Review). 4 (3): 102–8. PMC 4017418. PMID 24834166.CS1 maint: uses authors parameter (link)
  22. ↑ Tonutti, E; Bizzaro, N (2014). "Diagnosis and classification of celiac disease and gluten sensitivity". Autoimmun Rev (Review). 13 (4–5): 472–6. doi:10.1016/j.autrev.2014.01.043. PMID 24440147.CS1 maint: uses authors parameter (link)
  23. 1 2 3 Penagini, F; Dilillo, D; Meneghin, F; Mameli, C; Fabiano, V; Zuccotti, GV (November 2013). "Gluten-free diet in children: an approach to a nutritionally adequate and balanced diet". Nutrients (Review). 5 (11): 4553–65. doi:10.3390/nu5114553. PMC 3847748. PMID 24253052.CS1 maint: uses authors parameter (link)
  24. 1 2 Di Sabatino, A; Corazza, GR (April 2009). "Coeliac disease". Lancet. 373 (9673): 1480–93. doi:10.1016/S0140-6736 (09) 60254-3 Check |doi= value (help). PMID 19394538.CS1 maint: uses authors parameter (link)
  25. ↑ Pinto-Sánchez, MI; Causada-Calo, N; Bercik, P; Ford, AC; Murray, JA; Armstrong, D; Semrad, C; Kupfer, SS; Alaedini, A; Moayyedi, P; Leffler, DA; Verdú, EF; Green, P (August 2017). "Safety of Adding Oats to a Gluten-Free Diet for Patients With Celiac Disease: Systematic Review and Meta-analysis of Clinical and Observational Studies". Gastroenterology. 153 (2): 395-409.e3. doi:10.1053/j.gastro.2017.04.009. PMID 28431885.CS1 maint: uses authors parameter (link)
  26. ↑ Comino, I; Moreno, M; Sousa, C (November 2015). "Role of oats in celiac disease". World Journal of Gastroenterology. 21 (41): 11825–31. doi:10.3748/wjg.v21.i41.11825. PMC 4631980. PMID 26557006. It is necessary to consider that oats include many varieties, containing various amino acid sequences and showing different immunoreactivities associated with toxic prolamins. As a result, several studies have shown that the immunogenicity of oats varies depending on the cultivar consumed. Thus, it is essential to thoroughly study the variety of oats used in a food ingredient before including it in a gluten-free diet.CS1 maint: uses authors parameter (link)
  27. ↑ National Institute for Health and Clinical Excellence. Clinical guideline 86: Recognition and assessment of coeliac disease. London, 2015.
  28. ↑ Matthias, T; Pfeiffer, S; Selmi, C; M, Eric Gershwin (April 2010). "Diagnostic challenges in celiac disease and the role of the tissue transglutaminase-neo-epitope". Clin Rev Allergy Immunol (Review). 38 (2–3): 298–301. doi:10.1007/s12016-009-8160-z. PMID 19629760.CS1 maint: uses authors parameter (link)
  29. ↑ Lewis, NR; Scott, BB (July 2006). "Systematic review: the use of serology to exclude or diagnose coeliac disease (a comparison of the endomysial and tissue transglutaminase antibody tests)". Alimentary Pharmacology & Therapeutics. 24 (1): 47–54. doi:10.1111/j.1365-2036.2006.02967.x. PMID 16803602.CS1 maint: uses authors parameter (link)
  30. ↑ Rostom, A; Murray, JA; Kagnoff, MF (December 2006). "American Gastroenterological Association (AGA) Institute technical review on the diagnosis and management of celiac disease". Gastroenterology (Review). 131 (6): 1981–2002. doi:10.1053/j.gastro.2006.10.004. PMID 17087937.CS1 maint: uses authors parameter (link)
  31. ↑ Molina-Infante, J; Santolaria, S; Sanders, DS; Fernández-Bañares, F (May 2015). "Systematic review: noncoeliac gluten sensitivity". Alimentary Pharmacology & Therapeutics (Review). 41 (9): 807–20. doi:10.1111/apt.13155. PMID 25753138. Furthermore, seronegativity is more common in coeliac disease patients without villous atrophy (Marsh 1-2 lesions), but these ‘minor’ forms of coeliac disease may have similar clinical manifestations to those with villous atrophy and may show similar clinical-histological remission with reversal of haematological or biochemical disturbances on a gluten-free diet (GFD).CS1 maint: uses authors parameter (link)
  32. ↑ Ludvigsson, JF; Card, T; Ciclitira, PJ; Swift, GL; Nasr, I; Sanders, DS; Ciacci, C (April 2015). "Support for patients with celiac disease: A literature review". United European Gastroenterology Journal (Review). 3 (2): 146–59. doi:10.1177/2050640614562599. PMC 4406900. PMID 25922674.CS1 maint: uses authors parameter (link)
  33. ↑ van Heel, DA; West, J (July 2006). "Recent advances in coeliac disease". Gut (Review). 55 (7): 1037–46. doi:10.1136/gut.2005.075119. PMC 1856316. PMID 16766754.CS1 maint: uses authors parameter (link)
  34. ↑ Bibbins-Domingo, K; Grossman, DC; Curry, SJ; Barry, MJ; Davidson, KW; Doubeni, CA; Ebell, M; Epling, JW; Herzstein, J; Kemper, AR; Krist, AH; Kurth, AE; Landefeld, CS; Mangione, CM; Phipps, MG; Silverstein, M; Simon, MA; Tseng, CW (March 2017). "Screening for Celiac Disease: US Preventive Services Task Force Recommendation Statement". JAMA. 317 (12): 1252–1257. doi:10.1001/jama.2017.1462. PMID 28350936.CS1 maint: uses authors parameter (link)
  35. ↑ Lionetti, E; Gatti, S; Pulvirenti, A; Catassi, C (June 2015). "Celiac disease from a global perspective". Best Practice & Research. Clinical Gastroenterology (Review). 29 (3): 365–79. doi:10.1016/j.bpg.2015.05.004. PMID 26060103.CS1 maint: uses authors parameter (link)
  36. ↑ Hischenhuber, C; Crevel, R; Jarry, B; Mäki, M; Moneret-Vautrin, DA; Romano, A; Troncone, R; Ward, R (March 2006). "Review article: safe amounts of gluten for patients with wheat allergy or coeliac disease". Alimentary Pharmacology & Therapeutics. 23 (5): 559–75. doi:10.1111/j.1365-2036.2006.02768.x. PMID 16480395.CS1 maint: uses authors parameter (link)
  37. ↑ Adams F, translator (1856). "On The Cœliac Affection". The extant works of Aretaeus, The Cappadocian. London: Sydenham Society. pp. 350–1. สืบค้นเมื่อ 2009-12-12.
  38. ↑ Losowsky, MS (2008). "A history of coeliac disease". Digestive Diseases. 26 (2): 112–20. doi:10.1159/000116768. PMID 18431060.CS1 maint: uses authors parameter (link)

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